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Intro
Haglund's Deformity
is one component of the myriad of painful heel maladies. Commonly
referred as "the pump bump", Haglund's Deformity and its associated
soft tissue pathology may be the primary source of pain in the back
of the heel. The counter of the shoe further aggravates this area,
also known as the posterior heel. The patient and podiatric physician
can encounter much frustration trying to relieve the pain associated
with conservative care.
Anatomy
The Tendo-Achilles
(heel cord) inserts into the posterior middle one third surface
of the calcaneus (heel bone). Superior to this insertion site is
a bursal sac located between the Tendo-Achilles and the superior
one third of the posterior surface of the calcaneus. The bursal
sac functions as padding for this tendon and also provides protection
from the rigid surface of the calcaneus. Superficial to the Tendo-Achilles
is another bursal sac that develops from the pressure of shoegear.
This bursal sac is covered by the skin. Therefore, the Tendo-Achilles,
the strongest tendon in the human body, is further protected by
"sandwiching" the tendon between two bursal sacs. The superior surface
of the posterior calcaneus has a ridge of bone known as the bursal
projection. The entire surface of the superior portion of the calcaneus
to the posterior facet of the subtalar joint is concave. The area
that is superior to this part of the calcaneus is called Kager's
Triangle. Kager's Triangle is outlined by the top of the posterior
one half of the calcaneus as the base of the triangle. The Tendo-Achilles
and the posterior aspect of the tibia define the legs, or sides,
of this triangle.
Pathology
In the Haglund's
deformity, the bursal projection has marked osseous enlargement.
This enlargement (pump-bump) impinges upon the bursal sac and the
Tendo-Achilles. Furthermore, compression from the heel counter of
shoegear causes inflammatory reaction to occur. The skin will appear
reddened, boggy and/or painful during palpation. Pain may be felt
with dorsiflexion of the foot at the ankle joint. This is due to
the inflammatory component of this disorder.
Root et al
and Ruch in their articles have appropriately indicated the
function of calcaneus. For example, in a pes cavus foot, the calcaneus
has an increase in the calcaneal inclination angle causing the posterior-superior
calcaneus to be more impinged. Furthermore, forefoot varus, compensated
rearfoot varus and compensated forefoot valgus leads to abnormal
subtalar joint motion causing again increase prominence of the bursal
projection, thus pushing the pump-bump against the heel counter.
As the condition
of chronic pulling of a tendon at its insertion occurs, local inflammatory
conditions cause osseous spurring. Therefore, it is not uncommon
to find a posterior heel spur in this painful area. Therefore, the
development of an enlarged bursal sac, enlarged bursal projection
of the calcaneus and development of a posterior spur are usually
seen together.
Treatment
Conservative
care includes eliminating the heel counter causing pressure. This
is accomplished by a heel lift in the shoe or moving to a sling
back shoe. Resting the affected area with ice packs can help reduce
the inflammation. Anti-inflammatory medications like Aspirin or
Ibuprofen can slow the inflammatory response from worsening.
Steroid injections
should be avoided due to possible tendinopathy of the Tendo-Achilles.
However, a steroid used in an iontophoretic application may be of
benefit. In iontophoresis, a nine volt current is used to move (transduce)
the cationic structure of dexamethasone through the dermis to the
inflammed tissue, however, the concentration is low and administered
slowly.
Other conservative
care includes orthotic management to control the biomechanical deformity.
Physical therapy of whirlpool, contrast baths, ultrasound, and deep
massage may be employed.
Should the conservative
care fail, (some suggest 6 months or more of non-surgical care)
then surgical resection of the Haglund's deformity and the spur
if present. Often, the Tendo-Achilles will be partially or totally
detached to resect the spur. The tendon will be reattached with
tendon anchors. A non-weight bearing cast for 6-8 weeks is recommended.
Complications include failure to reduce pain, nerve entrapment of
the sural nerve, tendon dysfunction, delayed healing, infection
and reoccurrence of the deformity.
Dr.
Graham has been in private practice in Effingham since 1989 and
is on staff at St. Anthony's Memorial Hospital and Effingham Ambulatory
Surgical Treatment Center. He is Board Certified by the American
Board of Podiatric Surgery and American Board of Podiatric Orthopaedics
and Primary Podiatric Medicine. Dr. Graham is a Fellow of the American
College of Foot and Ankle Surgeons, the American College of Foot
and Ankle Orthopaedics and Medicine, and American Professional Wound
Care Association. He is also a member of the American Podiatric
Medical Association and the Illinois Podiatric Medical Association.
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