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Classically,
tendon injuries occur from an event of over-activity or repetitive
micro-insults that culminate into a painful syndrome that can lead
to a rupture of the tendon. The majority of these tendonopathies
involve those tendons which perform under great stress. The tendo-Achilles
and tibialis posterior tendons are the most commonly affected in
this array of tendon disorders.
The tendo-Achilles
is the largest tendon in the body. It is formed by three muscles,
the Gastrocnemius, the soleus, and the plantaris. These muscles
function to: provide heel lift during the gait cycle; plantar flex
the lateral column of the foot to the ground; decelerate the forward
momentum of the tibia (extending the knee); and cause external rotation
of the leg. The tendo-Achilles inserts into the posterior (back)
middle 1/3 of the calcaneus (heel bone). This insertion (attachment)
can be the source of problems. Bursal sacs located in this area
(between the bone and tendon, and between the tendon and the skin)
can become inflamed, resulting in Bursitis. The tendon can then
cause a spur formation secondary to the chronic inflammation.
The goal of
treating this disorder is to arrest the inflammation and irritation
to the tendon at its insertion. This can be achieved through the
use of non-steroidal anti-inflammatory medications, rest, heel lifts,
and protective measures, such as avoiding heel irritation with heel
cups. Orthotic devices may help with some heel tendonitis, and non-injected
steroids may also be utilized.
Another tendon
ailment is the "water-shed" injury. This is usually a
rupture at the least vascular area of the tendon. It is caused by
the foot being forced into the dorsiflexed position due to resistance
of the muscle. When this occurs, the patient will feel as if he
were hit in the leg with a baseball bat. He will not be able to
dorsiflex his foot, and his tendon will have an abnormal contour
with bruising. This particular injury requires a mandatory surgical
primary repair of the tendon. Some cases also require a harvest
graft of tendinous material from the plantaris or the tensor fascia
lata. Post op casting of 8-12 weeks is necessary to heel this major
disaster.
The tibialis
posterior tendon may also present with injury to its substance.
This may lead to dysfunction of the tendon. This muscle function
is to decelerate the pronation of the foot in the stance phase of
the gait cycle. Therefore, the tibialis posterior causes the arch
to rise as it supinates. It also decelerates the forward momentum
of the leg, which produces knee extension and heel lift as the foot
dorsiflexes at the ankle joint. The tibialis posterior tendon also
rotates the leg externally. Its activity begins just after heel
strike and continues after heel lift.
The patient
will complain of medial malleolus (ankle) pain at one of three sites:
behind, below, or at the insertion point of the medial (inside)
arch. These complaints include burning, aching, pulling, or ripping
sensations which are localized within the course of the tendon.
Pain is felt during the contact and stance phase of the gait cycle.
This pain, along with swelling, temperature, and redness, will increase
with use of the foot.
During tibialis
dysfunction the forefoot is abducted on the rearfoot, the heel is
everted, and the arch is collapsed in comparison to the contra lateral
limb. If the tendon is ruptured, the patient will be unable to invert
his heel on a toe raise. Also, when viewing from the back of the
heel, the examiner will see too many toes on the lateral side of
the foot, thus indicating the forefoot abduction. This foot will
have a collapsing arch that leads to other secondary complaints.
Assessing this
problem involves the collective use of x-rays, MRIs, tenograms,
and the clinical exam. Conservative care may render relief of tendonitis.
These measures consist of taping (strapping) the foot to relieve
the tendon, as well as the use of non-steroidal anti-inflammatory
medication, ice, arch devices (orthotics), and possible cast immobilization.
In the acute phase of this injury, avoiding activity (exercise)
of this muscle and its tendon is mandatory. If these measures fail
to arrest the problem, surgery is indicated.
The goal of
this surgery is to improve the function of the foot and eliminate
pain. The presence of an accessory ossicle (extra bone) in the tendon
at its insertion may cause the tendons function to weaken.
If this is the case, removal of the bone and advancement of the
tendon onto the navicular is in order. Pain from this particular
condition is usually located near the navicular bone.
If the pain
is located behind the medial malleolus, remodeling of this bone
is required due to the abnormal contour irritating the tendon. The
pain is localized here and the surgery is performed at this location.
As in the earlier case, a non-weight bearing cast is used 4-6 weeks
post operatively.
If the problem
of tibialis dysfunction is the case, then many procedures involving
tendon and osseous structures will be performed. The goal is addressing
the deforming forces of the tendon. The tibialis posterior tendon
will have a primary repair, as well as an anastomosis (joining)
of the flexor digitorum longus tendon. The calcaneus can either
be lengthened with a bone graft or rotated to invert. The tendo-Achilles
is usually lengthened. In the worst case scenario, fusion of joints,
such as a triple arthrodesis, may be needed. Remember it is the
foots inability to supinate that causes a strain on the tibialis
posterior tendon. The tibialis posterior tendon is thus secondarily
insulted. For 8-12 weeks post operatively, a non-weight bearing
cast is used.
Severe ankle
sprains involving inversion can disrupt the peroneal tendons and
can rupture, or more likely, cause a longitudinal split. Pain is
located both posterior and inferior to the fibular malleolus. Popping
and snapping are felt in range of motion. In this instance, the
peroneal longus is usually injured.
Diagnosis can
be confirmed with an arthrogram (x-ray of the ankle joint with radiopaque
dye). Spillage of the dye out of the joint on the lateral aspect
indicates tearing of the peroneal sheath and ligaments. Primary
surgical repair is in order, although conservative care should first
be employed. MRI studies can likewise reveal pathology of the tendon,
however, this is not 100% successful in visualizing the disorder.
Peroneal slippage
over the fibula may also symptomatically occur. The peroneal brevis
and longus tendons use the Fibula as a pulley to evert the forefoot.
This slippage can cause localized irritation in this area (posterior
fibular). Reconstruction of the Posterior lip of the fibula via
several surgical approaches should be utilized. Post operatively,
a non weight bearing cast for 4-6 weeks (minimum) is needed to protect
the heeling.
An argument
exists in the use of a steroid injection into the tendon sheath
to help heal the tendon. This approach should be used with extreme
caution. The limb is protected from the patient with the use of
a cast or a posterior splint for 2-4 weeks. It is not the steroid
itself, but the possible removal of protective pain by the steroid,
that can lead to a rupture.
Any tendon injury
can lead to either a partial or a total rupture of the substance.
This can be problematic for the surgeon. Total ruptures are similar
to a cable rupture, in that the ends are frayed. Grafting may help,
but the results are dependent on the health of the tendon and the
skill of the surgeon. Do not delay care for any tendinous injury.
Your results are dependent upon early treatment.
Abduction-
movement away from the midline of the body which divides into
right and left sides (Out-toeing)
Dorsiflexion-
movement of the top of the foot to the front of the tibia
Plantarflexion-
movement of the top of the foot away from the tibia
Pronation-
movement of lowering the arch to the ground
Supination-
movement of raising the arch from the ground
Adduction-
movement towards the midline of the body which divides into right
and left sides (In-toeing)
Medial-
Located near the midline of the body which divides into right
and left sides
Lateral-
Located away from the midline of the body which divides into right
and left sides
Dr.
Graham has been in private practice in Effingham since 1989 and
is on staff at St. Anthony's Memorial Hospital and Effingham Ambulatory
Surgical Treatment Center. He is Board Certified by the American
Board of Podiatric Surgery and American Board of Podiatric Orthopaedics
and Primary Podiatric Medicine. Dr. Graham is a Fellow of the American
College of Foot and Ankle Surgeons, the American College of Foot
and Ankle Orthopaedics and Medicine, and American Professional Wound
Care Association. He is also a member of the American Podiatric
Medical Association and the Illinois Podiatric Medical Association.
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